Treating trigeminal neuralgia with Biofeedback

Trigeminal neuralgia is a pain syndrome characterized by pain in one or more of the trigeminal nerve branches.


The trigeminal nerve, which has three branches – ophthalmic, upper jaw, and lower jaw – controls the muscles of chewing, lacrimal secretion, and salivary secretion. As a result, it controls the sensitivity of the teeth, oral cavity, upper jaw, sinuses, and almost all of the skin of the head and face.

Despite countless studies, the cause of the emergence of neuralgia has yet to be established.

Secondary neuralgia can be caused by a variety of trigeminal nerve-related conditions, including tumor lesions, aneurysms, arteriovenous malformations, and traumatic traumas.


The most noticeable sign is pain. The agony is intense, like a “knife strike” or an “electric shock.” It can occur in the upper, middle, or lower half of the face and is restricted to the trigeminal nerve territory, i.e. the maxillary level. The onslaught is brief, lasting between a few seconds to several minutes. The discomfort usually occurs on the right side, although it can sometimes radiate to other areas or generalize.

Pain comes unexpectedly, usually as a result of the activation of a stimulus, such as a light touch or light stimulation.


Trigeminal neuralgia is an uncommon illness characterized by unilateral paroxysmal pain in the distribution territory of one or more trigeminal nerve branches that can be provoked by innocuous stimuli. Trigeminal neuralgia, which was recently divided into three different groups by the European Academy of Neurology, must meet well-defined criteria specified by the International Classification of Headache Disorders (ICHD).


Patients with both idiopathic and classical trigeminal neuralgia are classified as having primitive trigeminal neuralgia. Two other phenotypes have been identified:

  • Trigeminal neuralgia with solely paroxysmal pain; 

  • Trigeminal neuralgia with concurrent chronic pain (patients with additional continuous pain between paroxysms with the same distribution and in the same period with paroxysmal pain).


Most of the time, the pain is unilateral; only 1-2% of the time, it is bilateral, but never simultaneously (affects one side then the other asynchronously). It is restricted to the trigeminal territory and is usually limited to one of the branches, but it can potentially spread to other branches. The maxillary branch is the most usually impacted, with discomfort radiating from the upper lip, nasal wing, and maxillary gum. The mandibular branch is the second most afflicted, with pain typically felt near the top of the chin, lower lip, and mandibular tooth region. The ophthalmic branch is implicated less frequently, and pain is felt in the supraorbital zone.


The most prevalent cause of primitive trigeminal neuralgia is contact with the trigger zone, a specific patch of skin, or mucosa. A person may have one or more trigger points, which are often found in the nasolabial groove of the upper lip in the case of maxillary neuralgia and the gingival/ alveolar edge of the mandible in the case of mandibular neuralgia. In persistent trigeminal neuralgia, trigger zones may overlap. Less frequently, the trigger zone lies outside the region of primitive trigeminal neuralgia, which is either another branch of the trigeminal nerve or the territory of C2-C3. The triggered pain has a latency that gradually increases around the painful territory and lasts for a particular amount of time after the stimulus is interrupted.

Temporospatial summation has also been observed in response to repeating stimuli with increasing intensity and extent of discomfort.


Patients should try to avoid an attack by applying pressure to the trigger area. They should also avoid any low-intensity mechanical stimulation of the trigger area that would normally be painless (washing, shaving, makeup, brushing teeth). Indirectly triggering painful attacks can be low temperatures, wind, or ordinary settings (talking, laughing, chewing, swallowing, shaving) enforcing avoidance behavior (frozen face). Patients try to be completely immobile by not talking or moving their lips at all, and they are scared of everyday actions like eating, washing their faces, and brushing their teeth, which become impossible. Sleep-related attacks are quite infrequent.


Following each painful episode, there is a refractory period in which the pain cannot be induced by a stimulus. Patients can use this refractory phase to practice gestures they are afraid of. Trigeminal neuralgia caused by multiple sclerosis is widespread in people between the ages of 40 and 50, is more common in women than males, and affects the right side more commonly than the left. The ophthalmic branch is usually afflicted, however other divisions of the trigeminal nerve might also be impacted. Neuralgia can be unilateral or bilateral, with 18% of MS patients reporting bilateral trigeminal neuralgia.


Sensitivity problems like hypoesthesia or anesthesia are common. However, their absence does not rule out the possibility of an underlying reason.


Quantum Biofeedback, more precisely Neurofeedback has been used to address a variety of chronic pain syndromes, including trigeminal neuralgia, migraine headaches, and complex regional pain syndrome. The biofeedback device (NUCLEUS or ED-X) tailors the required therapy to each patient’s specific condition and response to instruction. Neurofeedback training often entails increasing alpha and SMR activity while decreasing theta and beta activity in various regions of the cortex.

Quantum Biofeedback can detect virtually every type of pain.

The brain pathways involved in pain sensation overlap with the neural circuits implicated in depression. As a result, pain not only stimulates sensory portions of the brain, but also emotional centers, resulting in sadness, worry, and dread. Furthermore, depression activates brain areas associated with pain feeling.


You may experience severe pain as a result of this issue, which has been referred to as “the most agonizing pain known to humanity.”

Fortunately, cutting-edge medical innovations like Quantum Medical devices can help us comprehend where trigeminal neuralgia originates, how to halt or regulate it, and how to manage to maintain composure throughout this entire process.